The role of 17 beta hydroxysteroid dehydrogenases

Unlike activated or ameboid microglia, ramified microglia do not phagocytose cells and secrete fewer immunomolecules (including the MHC class I / II proteins). Microglia in this state are able to search for and identify immune threats while maintaining homeostasis in the CNS. [14] [26] [27] Although this is considered the resting state, microglia in this form are still extremely active in chemically surveying the environment. Ramified microglia can be transformed into the activated form at any time in response to injury or threat. [19]

Many studies have shown that it is possible to regenerate beta cells in vivo in some animal models. Research in mice studies have shown that beta cells can often regenerate to the original quantity number after the beta cells have undergone some sort of stress test, such as the intentional destruction of the beta cells in the mice subject or once the auto-immune response has concluded. While these studies have conclusive results in mice, beta cells in human subjects may not possess this same level of versatility. Investigation of beta cells following acute onset of Type 1 diabetes has shown little to no proliferation of newly synthesized beta cells, which suggests that the results seen in the mice models would not occur in human subjects either.

The role of 17 beta hydroxysteroid dehydrogenases

the role of 17 beta hydroxysteroid dehydrogenases

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