I totally understand what kind of job you had. I work at a Wally World distribution center. Started in shipping, loading about 3 to 4 full semi’s a day. Didn’t know how much weight I loaded. Then switched to non conveyable. When in dog food, I’d stack about 60k/lbs in 12 hours. That was MUCH easier than shipping. My first year, I struggled. Then I talked to my bro-in-law, who is a personal trainer, found and I started to do good. I was taking creatine and C4 prior to work, and took an Animal Pak with UniLiver every break, while eating protien every 3-4 hours. This brought me to Muscle for Life and The Books. I’m in maintenance department now, and are about to join a gym. I’ve been wanting to get the Legion multi’s and switch to Legion supplements. I’m about done reading BLS, and are gonna start the year one challenge. I’ve aready bought BBLS and Shredded Chef. I get excited every time I think about my goals.
Within those tissues where the SERM acts as an Estrogen antagonist, such as the hypothalamus and breast tissue, the SERM will bind to the Estrogen receptor and remain inert. Circulating Estrogen will be unable to bind to the Estrogen receptor, as it is occupied by the SERM. At the site of breast tissue, this results in the prevention (and possibly the decrease) of gynecomastia where Estrogen is the culprit. At the hypothalamus gland, GnRH release is achieved and ultimately results in increased Testosterone production as a result of the augmentation of the negative feedback loop in acting as an Estrogen antagonist at the pituitary gland  .
Because the ultimate goal of a steroid cycle is to increase strength and muscle size, the associated spike in estrogen which accompanies steroids such as Testosterone is considered undesirable. In order to disassociate the two effects, two classes of drug are used. Medications such as Nolvadex or Clomid target the estrogen receptors. They make it more difficult for the estrogen to exert it’s influence within the body thus allowing the testosterone to act more freely. The second class is aromatase inhibitors such as Femara. They target the aromatase enzyme itself in order to prevent the production of estrogen in the first place. Sometimes, it’s not always clear which option you should go with or even what the differences are between the two. Lets clear that up a little.